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Our results suggest that both increased S -glutathionylation processes and suppressed de-glutathionylation reactions contribute to hepatic S -glutathionylation in NAFLD
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Other studies discovered that mucositis, diarrhea, anemia, hepatotoxicity, or leukemia were more frequent in patients with the mutant genotype than in those with the wild-type [18,19,25,30,31,32]
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