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3e(1)], whereas its oxidative fate in mitochondria, as indicated by elevated levels of M + 2 and M + 4 oxaloacetic acid (OAA), was enhanced under all treatment conditions as compared to the Atm -WT counterparts [Fig
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doi: 10.1017/S0954422418000148 207 BattsonMLLeeDMJarrellDKHouSEctonKEWeirTLet al
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