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in 2022 (102) (data was taken from InsPECT database), where OR per tumor was of 93% and CR was 83.1%, though the findings vary when compared to the work of the same author, which concentrated on cutaneous SCC (103)
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It is assumed that dsRNA editing by ADAR could result in weakened recognition by the helicase domain of Dicer-2 which functions as a cytosolic sensor for dsRNA to trigger immune signalling, independent of dicer enzymatic activity (137) (see section 7.3)
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