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[6] Together, these findings suggest that Epithalon may influence longevity-related processes not only through telomerase and antioxidant pathways, but also through DNA regulation, immune signaling, and tissue-repair mechanisms
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Meanwhile, in our opinion, programmed cell death, particularly apoptosis, could be added to this axis because the mechanisms of apoptosis and other forms of programmed cell death are inextricably linked to the mitochondria, mTOR, and autophagy
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