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Wang et al., 2016[144])
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The virus leaves behind: T-cell exhaustion — immune cells that are present but functionally depleted Persistent viral antigen — fragments of SARS-CoV-2 that continue stimulating inflammation long after active infection has cleared11 Autoimmune activation — molecular mimicry between viral proteins and self-tissue triggering immune attacks on the body's own cells12 Thymosin Alpha-1 has been studied extensively as an immune modulator, particularly in viral disease contexts
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